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L-ascorbic acid induces apoptosis in human laryngeal epidermoid Hep-2 cells by modulating the nuclear factor kappa-light-chain-enhancer of activated B cells/mitogen-activated protein kinase/Akt signaling pathway

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Fig. 1. L-ascorbic acid (L-AA)-induced apoptotic cell death in Hep-2 cells. (A) Cells were incubated with different concentrations of L-AA for 24 hours or 48 hours, and the cell viability was determined via 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyl tetraz
Fig. 2. Induction of apoptosis via L-ascorbic acid (L-AA) in Hep-2 cells. (A) Histograms representing flow cytometry results of cells treated with 5 and 10 mM L-AA for 24 hours
Fig. 4. Caspase activation in L-ascorbic acid (L-AA)-treated Hep-2 cells. (A) The caspase-3, -8, and -9 activity was determined by treating the cells with 5 and 10 mM L-AA for 24 hours and the cell lysates were analyzed via a colorimetric assay

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