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COVID-19 Patients Upregulate Toll-like Receptor 4-mediated Inflammatory Signaling That Mimics Bacterial SepsisInfectious Diseases, Microbiology & Parasitology

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Fig. 1. Transcriptome analysis reveals that immune gene expression profiles of COVID-19 patients are distinct to HC
Fig. 2. COVID-19 infection boosts NF-κB signaling pathway. (A) The left (MILD) and right (SEVERE) sides of box represent the mean fold change in mRNA levels, compared  with HC
Fig. 3. Top 10 most significantly up- and down-regulated DEiGs in SEVERE patients. (A) Log2-transformed fold changes of 579 immune-genes from MILD vs
Fig. 4. Recombinant NC and S2 ECD proteins of SARS-CoV2 robustly induce the expression of pro-inflammatory cytokines/chemokines in human primary PBMCs

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