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Treatment for NystagmusSeong-Hae Jeong

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Treatment for Nystagmus

Seong-Hae Jeong

Department of Neurology, Chungnam National University Hospital, Daejeon, Korea

Treatment for Nystagmus

Seong-Hae Jeong Chungnam National University Hospital

Goals of Treatment

Clear vision

Visual fixation

Eccentric

gaze-

holding

mechanism

VOR

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Clin Neuroophthalmol 6(Suppl):S27-34, April 2016 S28 http://neuro-ophthalmology.co.kr

Physiologic movement eye

Nystagmus slow-phase

speed

Tenotomy or botulinum toxin

Not require specific Treatment

 Infantile nystagmus

 Peripheral vestibular nystagmus

 Drug intoxication

General Approaches to Treatment

medical optical surgical

Acquired

Downbeat nystagmus

Upbeat nystagmus

Torsional nystagmus

Gaze-evoked nystagmus

Seesaw nystagmus

Periodic alternating nystagmus

Acquired pendular nystagmus in MS

Acquired pendular nystagmus in OPT

Congenial

 Infantile nystagmus syndrome

 Other congenital nystagmus

Acquired

Downbeat nystagmus

Upbeat nystagmus

Torsional nystagmus

Gaze-evoked nystagmus

Seesaw nystagmus

Periodic alternating nystagmus

Acquired pendular nystagmus in MS

Acquired pendular nystagmus in OPT

Congenial

 Infantile nystagmus syndrome

 Other congenital nystagmus

Downbeat nystagmus

 Acquired cerebellar degenerations

 Stroke

 Hindbrain anomalies (e.g., Chiari malformation)

 Multiple sclerosis

 Medications(lithium, anticonvulsants, opoioids)

 Idiopathic

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HIRSCHBERG’S TEST: CORNEAL LIGHT REFLEX

Rough measure of deviation

Note location of corneal light reflex

1mm = 7°, or 15 ∆

(4)

Clin Neuroophthalmol 6(Suppl):S27-34, April 2016 S30 http://neuro-ophthalmology.co.kr

1. Involvement of excitatory burst neurons in the PPRF

: SCA2 (Horn AKE et al, Neuro-ophthalmology 1996;16:229-240) Experimental slow saccades by injecting lidocaine into the PPRF (Barton et al., 2003)

2. Lesions of the omnipuase neurons

: using experimental excitotoxins are also reported to cause slow horizontal and vertical saccades. (Kaneko CRS, J Neurophysiol 1996;75:2229-2242) 3. Both burst (PPRF) and omnipause cell populations may be affected : slow saccade after cardiac surgery (Hanson, et al. Ann Neurol 1986;20;209-217)

4. It is also possible that disturbance of other inputs to the posterior burst

neurons (i.e., LLBN) could lead to slow saccades. For example, acute

inactivation of the superior colliculus causes slow saccades in monkey.

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Upbeat nystagmus

 Baclofen

JNNP 1991

burst neurons for vertical and torsional saccades

: vertical saccadic paresis

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Clin Neuroophthalmol 6(Suppl):S27-34, April 2016 S32 http://neuro-ophthalmology.co.kr

Gaze-Evoked Nystagmus

 Because gaze-evoked nystagmus does not usually produce visual symptoms, it does not need specific treatment.

Acquired pendular Nystagmus

Oculopalatal tremor

 Mefloquine

Proc Natl Acad Sci USA 2004

 Clonazepam

Neurology 2006

 Gabapentin and Memantine

Ann Neurol 2010

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Ann Neurol 2010

and reduced visual acuity. During treatment with gabapentin 1,200 mg/day, there was substantial decrease in his nystagmus and his visual acuity improved (Video 11–18). (B) Axial T2-weighted magnetic resonance imaging (MRI) showed an old pontine tegmental infarction (left panel; arrowhead) and hypertrophy of the right inferior olivary nucleus (indicated by arrowhead).

Chapter: Acquired Pendular Nystagmus and its Relationship to Visual Pathways Author(s): R. John Leigh and David S. Zee

From: The Neurology of Eye Movements (5 ed.)

Downloaded from Oxford Medicine Online. © Oxford University Press, 2015

Seesaw and hemiseesaw nystagmus

 Pendular seesaw nystagmus: alcohol or clonazepam

Rev Neurol 1995

 Mematine

Ann Neurol 2010

Treatment of nystagmus in EA2

 Acetazolamide

Neurology 1978

 4-aminopyridone Neurology 2011

 Dalfampridine

 Flunarizine

Eur Neurol 2006

Treatment of Congenital nystagmus Treatment of Congenital nystagmus

 Channelopathy

Semin Ophthalmol. 2006; 21:83–86

 Oral acetazolamide

Open Ophthalmol J 2010

 Topical brinzolamide

JNO 2011

 Gene therapy – Leber’s congenital amaurosis

NEJM 2009

http://bcove.me/53hssnmr

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Clin Neuroophthalmol 6(Suppl):S27-34, April 2016 S34 http://neuro-ophthalmology.co.kr

Treatment of Saccadic Intrusions and Oscillations

 Memantine

 Benzodiazepines

Treatments for ocular flutter and opsoclonus

 Propranolol

 Clonazepam

 Gabapentin

 Topiramate

 IVIG

 Corticosteroids

 ACTH

 Rituximab

 Ofatumumab

Optical treatments of abnormal eye movements

http://bcove.me/in111er2

Procedures to Weaken in EOM

Application of somatosensory or Auditory stimuli to suppress nystagmus

Figure 11–27 Effects of vibration at 100 Hz over the left mastoid process on monocular nystagmus in a patient with multiple sclerosis (Video 11–17). The nystagmus immediately decreases in amplitude after the stimulus is applied and continues to decrease during the stimulation period (gray area), increasing promptly when the stimulus is stopped.

Chapter: Treatments for Nystagmus and Saccadic Intrusions Author(s): R. John Leigh and David S. Zee From: The Neurology of Eye Movements (5 ed.) Downloaded from Oxford Medicine Online.

Reproduced with permission from Beh

SC, Tehrani AS, Kheradmand A, Zee DS.

Damping of monocular pendular nystagmus with vibration in a patient with multiple sclerosis.

Neurology. 2014; 821: 380–381.

http://bcove.me/bhlhty8z

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