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Personalized Medicine in Coronary Artery Disease: Insights From Genomic Research

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Fig. 1. Variants with different frequencies, penetrance, and dis- dis-ease susceptibility
Table 2. Candidate gene variation for atherothrombotic cardiovas- cardiovas-cular disease reported in recent meta-analyses of association  studies
Table 3. Susceptibility loci identified in four genome-wide asso- asso-ciation studies of CAD
Fig. 2. Biological pathways implicated in CAD pathophysiology and examples of genes. PCSK9 : proprotein convertase subtilisin/kexin 9,  APOA5 :  apoliprotein A-V, USF1 :  upstream transcription factor 1, CAD: coronary artery disease,  FLAP :  5’-lipoxygena
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