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Toll-like receptor 4 in lymphatic endothelial cells contributes to LPS-induced lymphangiogenesis by chemotactic recruitment of macrophages

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Figure 3. TLR4 in LECs is a functional receptor mediating LPS- LPS-induced NF- ␬B signaling
Figure 7. Schematic diagram on the role of TLR4 in LECs in the context of LPS-induced inflammation, resulting in lymphangiogenesis by chemotactic migration of macrophages that produce VEGF-C and VEGF-D

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